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Discover the signs, understand the disease, and learn how to save your cat’s life.
Understanding
FIP & Its Signs
Signs of FIP can arise weeks, months, or even years after initial infection. Early on, a cat may be asymptomatic or show vague signs:
Stunted growth / growth retardation.
Increased susceptibility to common infections.
Note: Early upper respiratory signs are usually caused by secondary pathogens (herpesvirus, mycoplasma, etc.), not directly by the FIP virus itself.
When FIP transitions into the clinical stage, cats often show general symptoms that are resistant to standard antibiotics:
Fluctuating, antibiotic-unresponsive fever.
Loss of appetite (Anorexia) & weight loss.
Lethargy and depression.
Rough, unkempt coat.
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The Different Forms of FIP
FIP Disease Forms Summary
Effusive (Wet) FIP High-protein fluid leakage due to blood vessel inflammation (Vasculitis).
Primary Characteristics & Symptoms:
- Ascites: Progressive, non-painful abdominal distension due to fluid accumulation.
- Dyspnea: Labored breathing or exercise intolerance caused by fluid gathering in the pleural space around the lungs.
Non-Effusive (Dry) FIP Localized inflammatory masses (granulomas) inside vital internal organs.
Primary Characteristics & Symptoms:
- Organ Damage: Directly attacks and affects the kidneys, spleen, liver, and terminal bowel.
- High Risk: Accounts for over 50% of inflammatory eye and nervous system diseases in cats under 3–5 years of age.
Ocular FIP The main presenting sign in 14% of dry FIP cases (and up to 70% in clinical combinations).
Primary Characteristics & Symptoms:
- Anterior Uveitis: Severe intraocular inflammation making the eyes look cloudy.
- Iris Changes: Notable and abnormal modifications in the color of the iris.
- Vascular Damage: Presence of flocculant debris in the eye chamber, retinal hemorrhages, or full retinal detachment.
Neurological FIP Occurs in 22% of dry cases when the virus escapes past the blood-to-brain barrier.
Primary Characteristics & Symptoms:
- Early / Vague Signs: Behavioral updates including dementia, aggressive behavior, compulsive licking, reluctance to jump, or muscle twitching.
- Advanced / Critical Signs: Ataxia (uncoordinated walking), progressive hindleg paralysis, seizures, hyperesthesia (oversensitivity to touch/sound), and fecal or urinary incontinence.
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Diagnosis & Confirmatory Tests
A definitive diagnosis is reached by compiling clinical history, thorough physical examinations, and specific structural and fluid laboratory diagnostics.
Bloodwork Checklist
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RBC / HCT (Red Blood Cells)
Often low, indicating non-regenerative anemia. An HCT level below 15% is highly critical and requires urgent attention.
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WBC / Neutrophils
Typically elevated significantly, acting as a clear biological response to active tissue inflammation.
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Lymphocytes
Often low (Lymphopenia), reflecting a compromised or exhausted systemic cell-mediated immune response.
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Proteins & A:G Ratio
Characterized by elevated Globulin (inflammatory marker) and decreased Albumin. Ideally, we want the overall A:G ratio to rise toward 0.7 or higher during treatment progress.
CSF & Aqueous Humor Analysis
Cerebrospinal Fluid (brain/spine) and aqueous humor (eye) fluid panels reveal highly elevated protein levels and high nucleated cell counts, predominantly dominated by neutrophils.
Advanced RT-PCR Testing
Highly sensitive and specific when utilizing laboratory assays that target the FCoV 7b gene on extracted body fluid or affected tissue specimens.
Diagnostic MRI Imaging
Neurological assessments reveal vital structural modifications including marked meningeal contrast enhancement, ventriculomegaly (hydrocephalus), or cerebral swelling.
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